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Low expression of GLP-1R occurred in parietal cells and smooth muscle cells of the stomach and in myenteric plexus neurons of the gut with no expression in the liver and thyroid gland [392]

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The improved glucose metabolism is mainly due to three closely interacting factors: 1) rapid absorption of glucose from the intestine giving rise to high post-prandial glucose responses, 2) a consequent exaggerated secretion of GLP-1 that act on the β-cells, and 3) radically improved hepatic and subsequently peripheral insulin sensitivity [226]