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In STZ-treatment rats, Exendin-4 improved learning and memory performance, protected hippocampal neurons against degeneration, and reversed STZ-induced tau hyperphosphorylation through downregulation of GSK-3β activity (154, 155, 182)
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But the current evidence is limited: While GLP-1s may be linked to improvements in factors that drive MS disease activity, these are associations — there’s no clear proof that GLP-1s directly slow MS progression, she cautions
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